Potent and noncompetitive inhibitor of both glutaminase GLS1 splice variants (KGA and GAC, IC 50 ~ 25 nM), but not GLS2. It limits the entry of glutamine derivates into the TCA cycle, starves tumor cells of glutamine, and ablates stem-like cells in GLS-overexpressing glioblastoma cells....
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Potent and noncompetitive inhibitor of both glutaminase GLS1 splice variants (KGA and GAC, IC 50 ~ 25 nM), but not GLS2. It limits the entry of glutamine derivates into the TCA cycle, starves tumor cells of glutamine, and ablates stem-like cells in GLS-overexpressing glioblastoma cells. Effective in a variety of cancer models, including triple-negative breast cancer and squamous cell carcinoma
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