A novel tool that specifically interacts with cryptochrome (CRY) preventing its ubiquitin-dependent degradation resulting in lengthening of the circadian period. KL-001-mediated CRY stabilization inhibits glucagon-induced gluconeogenesis in primary hepatocytes. Binds to the FAD-binding pocket of...
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A novel tool that specifically interacts with cryptochrome (CRY) preventing its ubiquitin-dependent degradation resulting in lengthening of the circadian period. KL-001-mediated CRY stabilization inhibits glucagon-induced gluconeogenesis in primary hepatocytes. Binds to the FAD-binding pocket of CRY2 as determined by co-crystal structure. Induces an increase in period along with simultaneous reduction in amplitude of circadian reporter expression in mammalian cells
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