Anti-Mouse CCL20/MIP-3-alpha Antibody (SAA2679) from ProteoGenix; catalogue ARO-A23116; research use only.
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The Anti-Mouse CCL20/MIP-3-alpha Antibody (SAA2679) is a highly specific and reliable tool for biotech research. This antibody targets the CCL20/MIP-3-alpha protein, also known as macrophage inflammatory protein 3-alpha, which plays a crucial role in immune response and inflammation.
The biological function of CCL20/MIP-3-alpha is to attract immune cells, specifically dendritic cells and T cells, to sites of inflammation or infection. This process is essential for the body’s defense against pathogens and plays a significant role in the development of autoimmune diseases.
The Anti-Mouse CCL20/MIP-3-alpha Antibody (SAA2679) is widely used in various applications, including immunohistochemistry (IHC), western blotting, and flow cytometry. It has been validated for use in mouse models, making it an ideal choice for preclinical studies.
In IHC, this antibody can be used to detect the expression of CCL20/MIP-3-alpha in tissue samples, providing valuable insights into the role of this protein in various diseases. In western blotting, it can detect the presence of CCL20/MIP-3-alpha in cell lysates, helping researchers understand its expression and regulation in different cell types.
Flow cytometry with the Anti-Mouse CCL20/MIP-3-alpha Antibody (SAA2679) allows for the identification and quantification of CCL20/MIP-3-alpha-expressing cells in complex cell populations. This is particularly useful for studying the recruitment and activation of immune cells in response to inflammation.
Researchers have successfully used this antibody in various experimental settings, including studies on inflammatory bowel disease, rheumatoid arthritis, and cancer. Its high specificity and sensitivity make it a valuable tool for understanding the role of CCL20/MIP-3-alpha in these and other diseases.
In summary, the Anti-Mouse CCL20/MIP-3-alpha Antibody (SAA2679) is a powerful tool for biotech research, enabling the detection and quantification of CCL20/MIP-3-alpha in various experimental settings. Its high specificity and reliability make it an essential component of any immunology or inflammation research project. Order now and take your research to the next level!
ProteoGenix catalogue number ARO-A23116. For research use only.
Target: Beta-chemokine exodus-1, C-C motif chemokine 20, CC chemokine LARC, CC chemokine ST38, Ccl20, Larc, Liver and activation-regulated chemokine, MIP-3-alpha, Macrophage inflammatory protein 3 alpha, Scya20, Small-inducible cytokine A20; Clonality: Monoclonal Antibody; Reactivity: Mouse; Applications: ELISA, Neutralization.
Available configurations: 50ug, 100ug, 1mg.
Fully tested in-house by ABMIUM, or supported by an advanced validation report from a partner reviewed by ABMIUM. Highest confidence. Non-conformities fully supported under the ABMIUM Product Promise.
Quality verified. Independent collaborator or expected-performance data available. Product meets ABMIUM quality standards.
Expected to work based on structural and biochemical data. Not yet directly tested by ABMIUM or a collaborator. No non-conformity support for this combination.
Not recommended for this application or species/sample combination. This may indicate either evidence of unsuitability or that the combination has not been tested and therefore cannot currently be recommended.
Evidence status by application and model.
| Species | WB | IHC-P | IHC-Fr | IF/ICC | Flow Cyt | IP | ChIP | ELISA | Neutralization |
|---|---|---|---|---|---|---|---|---|---|
| Mouse |
This product is manufactured by ProteoGenix and sold through ABMIUM without relabelling.
ABMIUM supports global orders. Availability, shipping requirements and applicable import arrangements are confirmed before fulfilment where required.
Yes. Send your target, sample, application and experimental conditions through ABMIUM MATCH or technical support.
Unless the listing expressly states otherwise, ABMIUM products are supplied for research use only and are not for diagnostic or therapeutic use.
