Anti-Mouse AHSG Antibody [B405] with collaborator-tested evidence for IHC-P.
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Anti-Mouse AHSG Antibody [B405] is a research-use antibody from Elabscience for studying AHSG in IHC-P workflows with listed reactivity for Mouse. Specifications and supporting documents below should be reviewed when planning the experiment.
Fully tested in-house by ABMIUM. Highest confidence. Non-conformities fully supported under the ABMIUM Product Promise.
Quality verified. Independent collaborator or expected-performance data available. Product meets ABMIUM quality standards.
Expected to work based on structural and biochemical data. Not yet directly tested by ABMIUM or a collaborator. No non-conformity support for this combination.
Not recommended for this application or species/sample combination. This may indicate either evidence of unsuitability or that the combination has not been tested and therefore cannot currently be recommended.
Evidence status by application and model.
| Species | WB | IHC-P | IHC-Fr | IF/ICC | Flow Cyt | IP | ChIP |
|---|---|---|---|---|---|---|---|
| Mouse |
This product is manufactured by Elabscience and sold through ABMIUM without relabelling.
ABMIUM supports global orders. Availability, shipping requirements and applicable import arrangements are confirmed before fulfilment where required.
Yes. Send your target, sample, application and experimental conditions through ABMIUM MATCH or technical support.
Unless the listing expressly states otherwise, ABMIUM products are supplied for research use only and are not for diagnostic or therapeutic use.
AHSG encodes alpha-2-HS-glycoprotein (fetuin-A), a secreted serum glycoprotein synthesized by hepatocytes and concentrated in bone matrix. The AHSG/fetuin-A target is studied in mineral handling, calcification, bone biology and metabolic or inflammatory physiology.
Biological function: Alpha-2-HS-glycoprotein (AHSG; fetuin-A) is a secreted plasma glycoprotein with calcium-binding and opsonic properties. Reviewed UniProt annotations support roles in endocytosis, influence on the mineral phase of bone and regulation of bone mineralization.
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