ATF1 RUO break-apart FISH probe reagent with locus, fluorophore, specimen, protocol and handling fields mapped.
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Purpose: ATF1 Break Apart FISH Probe Reagent is a Research Use Only FISH probe reagent for research assessment of rearrangements involving ATF1.
Probe design: Break-apart design; target/locus ATF1 Break Apart.
Format: 100µL/10 Tests.
Fully tested in-house by ABMIUM. Highest confidence. Non-conformities fully supported under the ABMIUM Product Promise.
Quality verified. Independent collaborator or expected-performance data available. Product meets ABMIUM quality standards.
Expected to work based on structural and biochemical data. Not yet directly tested by ABMIUM or a collaborator. No non-conformity support for this combination.
Not recommended for this application or species/sample combination. This may indicate either evidence of unsuitability or that the combination has not been tested and therefore cannot currently be recommended.
Sample compatibility evidence using the same validation key.
| Sample type | Compatible |
|---|---|
| Prepared FISH slide |
This product is manufactured by Wuhan HealthCare Biotechnology and sold through ABMIUM without relabelling.
ABMIUM supports global orders. Availability, shipping requirements and applicable import arrangements are confirmed before fulfilment where required.
Yes. Send your target, sample, application and experimental conditions through ABMIUM MATCH or technical support.
Unless the listing expressly states otherwise, ABMIUM products are supplied for research use only and are not for diagnostic or therapeutic use.
Activating transcription factor 1 (ATF1), reviewed by UniProt as cyclic AMP-dependent transcription factor ATF-1, is a bZIP transcription factor that links cAMP/PKA signalling to CRE-dependent gene regulation. Human ATF1 maps to NCBI Gene 466 and reviewed UniProtKB P18846; this record links the verified target to related ABMIUM catalogue products.
Biological function: ATF1 binds cAMP response elements (CREs) in promoters and mediates PKA-responsive transcription. Phosphorylation within its kinase-inducible domain enhances transactivation, supporting stimulus-responsive regulation of gene expression.
